Abstract BACKGROUND Maintenance of kidney function in kidney allografts remains a challenge, as the allograft often progressively develops fibrosis after kidney transplantation. Fibrosis is caused by the accumulation of extracellular matrix proteins like type I and III collagen (COL I and III) that replace the functional tissue. We assessed the concentrations of a neo-epitope fragment […]
A non-invasive biomarker of type III collagen degradation reflects ischaemia reperfusion injury in rats.
August 1, 2019
Nephrol Dial Transplant